Selected article for: "present study and tumor macrophage"

Author: Park, Jeong-In; Song, Kyung-Hee; Jung, Seung-Youn; Ahn, Jiyeon; Hwang, Sang-Gu; Kim, Joon; Kim, Eun Ho; Song, Jie-Young
Title: Tumor-Treating Fields Induce RAW264.7 Macrophage Activation Via NK-?B/MAPK Signaling Pathways
  • Document date: 2019_8_11
  • ID: 65s65ojc_33
    Snippet: Some of these key cytokines that play critical roles in immune and inflammatory processes include TNF-a, which is also produced by T-lymphocytes and natural killer cells, and IL-1b, which facilitates the release of activated macrophages. 35, 36 In the present study, TTFs markedly treated RAW 264.7 cells to release TNF-a and IL-1b, indicating that TTFs-stimulated macrophages contribute to provoking an inflammatory response. The levels of TNF-a, IL.....
    Document: Some of these key cytokines that play critical roles in immune and inflammatory processes include TNF-a, which is also produced by T-lymphocytes and natural killer cells, and IL-1b, which facilitates the release of activated macrophages. 35, 36 In the present study, TTFs markedly treated RAW 264.7 cells to release TNF-a and IL-1b, indicating that TTFs-stimulated macrophages contribute to provoking an inflammatory response. The levels of TNF-a, IL-1b, and IL-6 were markedly higher in TTFs-treated RAW 264.7 cells cocultured with 4T1 murine mammary carcinoma cells than in 4T1 or RAW 264.7 cells with or without TTFs. Moreover, the viability of 4T1 cells treated with the CM of TTFs-stimulated RAW 264.7 cells decreased, indicating that macrophage activation by TTFs effectively killed the tumor cells.

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