Author: Wang, Ziqiang; Zhao, Yiwan; Zhang, Yaou
Title: Viral lncRNA: A regulatory molecule for controlling virus life cycle Document date: 2017_3_23
ID: 213ddk0s_20
Snippet: Moreover, virus-infected cells may initiate apoptosis to prevent the spread of infection to other cells. As a countermeasure, human cytomegalovirus encodes Beta 2.7, which is the most abundantly transcribed early gene, to prevent the host cell stress response and apoptosis by binding to components of the mitochondrial respiratory chain complex; (reduced nicotinamide adenine dinucleotideubiquinone oxido-reductase). This interaction is also importa.....
Document: Moreover, virus-infected cells may initiate apoptosis to prevent the spread of infection to other cells. As a countermeasure, human cytomegalovirus encodes Beta 2.7, which is the most abundantly transcribed early gene, to prevent the host cell stress response and apoptosis by binding to components of the mitochondrial respiratory chain complex; (reduced nicotinamide adenine dinucleotideubiquinone oxido-reductase). This interaction is also important for the stabilization of the mitochondrial membrane potential and mitochondrial ATP production and the prevention of metabolic dysfunction, which are essential for the completion of the viral life cycle [59] . The psoriasis susceptibility-related RNA gene induced by stress (PRINS) is a lncRNA overexpressed by herpes simplex viral infection [60] ; upregulated PRINS can decrease cell apoptosis by regulating the anti-apoptotic G1P3 protein [61] .
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