Author: Lee, Yun Jin
Title: Acute disseminated encephalomyelitis in children: differential diagnosis from multiple sclerosis on the basis of clinical course Document date: 2011_6_30
ID: zroskqmk_4
Snippet: The pathogenesis of ADEM is incompletely understood. The proposed mechanism of ADEM is that myelin autoantigens, such as myelin basic protein, proteolipid protein, and myelin oligodendrocyte protein, share antigenic determinants with those of an infecting pathogen 12) . This model is supported by studies of lymphocytes in children with ADEM. In 1 report, the frequency of T cell reactivity to myelin basic protein was 10 times higher in patients wi.....
Document: The pathogenesis of ADEM is incompletely understood. The proposed mechanism of ADEM is that myelin autoantigens, such as myelin basic protein, proteolipid protein, and myelin oligodendrocyte protein, share antigenic determinants with those of an infecting pathogen 12) . This model is supported by studies of lymphocytes in children with ADEM. In 1 report, the frequency of T cell reactivity to myelin basic protein was 10 times higher in patients with ADEM than in those with encephalitis or normal controls 5) .
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