Author: Glennie, S; Gritzfeld, J F; Pennington, S H; Garner-Jones, M; Coombes, N; Hopkins, M J; Vadesilho, C F; Miyaji, E N; Wang, D; Wright, A D; Collins, A M; Gordon, S B; Ferreira, D M
Title: Modulation of nasopharyngeal innate defenses by viral coinfection predisposes individuals to experimental pneumococcal carriage Document date: 2015_4_29
ID: st475jw7_10
Snippet: We used a model of noninflamed and inflamed nasopharyngeal epithelium to investigate the role of mucosal FH in pneumococcal adherence during health and coinfection, respectively. Inflammation of epithelium was confirmed by increased expression of polymeric immunoglobulin receptor and the receptor for platelet-activating factor (data not shown). We observed more than twofold increase in pneumococcal adherence of D39 to inflamed epithelium compared.....
Document: We used a model of noninflamed and inflamed nasopharyngeal epithelium to investigate the role of mucosal FH in pneumococcal adherence during health and coinfection, respectively. Inflammation of epithelium was confirmed by increased expression of polymeric immunoglobulin receptor and the receptor for platelet-activating factor (data not shown). We observed more than twofold increase in pneumococcal adherence of D39 to inflamed epithelium compared with noninflamed epithelium (Figure 3a , black squares).
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