Author: Tan, Zhaoli; Gao, Lihua; Wang, Yan; Yin, Huihui; Xi, Yongyi; Wu, Xiaojie; Shao, Yong; Qiu, Weiyi; Du, Peng; Shen, Wenlong; Fu, Ling; Jia, Ru; Zhao, Chuanhua; Zhang, Yun; Zhao, Zhihu; Sun, Zhiwei; Chen, Hongxing; Hu, Xianwen; Xu, Jianming; Wang, Youliang
Title: PRSS contributes to cetuximab resistance in colorectal cancer Document date: 2020_1_1
ID: tymoeyoo_80
Snippet: We first downloaded the PRSS1 and PRSS3 expression files and clinical files for all 38 cancer types from TCGA (https://tcga-data. nci.nih.gov/docs/publications/stad_2014/). The clinical files and expression results were obtained from level 3 TCGA data. On the basis of the median PRSS1 or PRSS3 level of the cancer types in the TCGA database, we divided the patients who received cetuximab monotherapy or bevacizumab monotherapy into high-and low-PRS.....
Document: We first downloaded the PRSS1 and PRSS3 expression files and clinical files for all 38 cancer types from TCGA (https://tcga-data. nci.nih.gov/docs/publications/stad_2014/). The clinical files and expression results were obtained from level 3 TCGA data. On the basis of the median PRSS1 or PRSS3 level of the cancer types in the TCGA database, we divided the patients who received cetuximab monotherapy or bevacizumab monotherapy into high-and low-PRSS1 or high-and low-PRSS3 groups. Kaplan-Meier methods were used to estimate PFS and OS.
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