Research Communications of the 27(th) ECVIM-CA Congress: Intercontinental, Saint Julian's, Malta, 14th to 16th September 2017

From February 2015 to December 2015, 16 cats were recruited. Seven cats completed the study (stress precluded adequate blood sampling in six cats, one cat removed the feeding tube before day 5, two cats died). The seven cats were 9.3 AE 3.8 years old. Six cats were anorectic from three to seven days, one cat was dysorexic for three months. Two cats with an hepatic lipidosis were considered at risk to develop a refeeding syndrome. One of them with a threemonth history of dysorexia, icterus and marked emaciation at admission developed at day 3 of refeeding severe electrolyte shifts (marked hypokalemia and hypomagnesemia, moderate hypophosphatemia), aggravation of anemia, acute elevation of creatine kinase activity and bilirubinemia concomitantly with respiratory distress, vomiting, muscle weakness and hypotension. He was strongly suspected of refeeding syndrome. None of the six other cats developed clinical signs or remarkable biological abnormalities potentially linked with refeeding. Glycemia and insulinemia were consistently higher on day 3 and 5 than on day 0 on all cats. Mild to moderate hypophosphatemia was documented in 4/6 cats, mild hypokalemia in 2/6 cats and mild hypomagnesemia in 1/6 cats.