Selected article for: "cell cell and skeletal muscle"
Author: Xie, Xin-Hui; Wang, Xin-Luan; Yang, Hui-Lin; Zhao, De-Wei; Qin, Ling
Title: Steroid-associated osteonecrosis: Epidemiology, pathophysiology, animal model, prevention, and potential treatments (an overview)
  • Document date: 2015_1_13
    • ID: y6y235hw_57
    Snippet: Prevention of glucocorticoid-induced apoptosis in osteocytes and osteoblasts might be a potential method to prevent the development of ON. Calcium-binding protein calbindin-D28k, and granulocyte colony stimulating factor and stem cell factor have been reported to prevent glucocorticoid-induced bone cell death through inhibition of glucocorticoid-induced caspase 3 activation, as well as ERKs activation [59, 60] . Bisphosphonates and calcitonin pro.....
    Document: Prevention of glucocorticoid-induced apoptosis in osteocytes and osteoblasts might be a potential method to prevent the development of ON. Calcium-binding protein calbindin-D28k, and granulocyte colony stimulating factor and stem cell factor have been reported to prevent glucocorticoid-induced bone cell death through inhibition of glucocorticoid-induced caspase 3 activation, as well as ERKs activation [59, 60] . Bisphosphonates and calcitonin promoted a rapid increase in the phosphorylated fraction of ERKs and inhibited the apoptosis of osteocytes and osteoblasts following prednisolone administration to mice. Recently, the skeletal muscle secreted factors were found to prevent glucocorticoid-induced osteocyte apoptosis through activation of b-catenin.

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