Selected article for: "antiviral response and cell infection"

Author: Madera, Sharline; Rapp, Moritz; Firth, Matthew A.; Beilke, Joshua N.; Lanier, Lewis L.; Sun, Joseph C.
Title: Type I IFN promotes NK cell expansion during viral infection by protecting NK cells against fratricide
  • Document date: 2016_2_8
  • ID: qkdni38b_3
    Snippet: Type I IFNs signal through STAT1-STAT2 heterodimers and STAT1-STAT1 homodimers (Li et al., 1996) . Therefore, we determined the role of STAT1 in the NK cell response to MCMV infection using STAT1-deficient mice. Equal numbers of WT and Stat1 −/− Ly49H + NK cells were cotransferred into Ly49H-deficient hosts and then infected with MCMV. Similar to Ifnar −/− NK cells, Stat1 −/− NK cells exhibited a striking defect in expansion during th.....
    Document: Type I IFNs signal through STAT1-STAT2 heterodimers and STAT1-STAT1 homodimers (Li et al., 1996) . Therefore, we determined the role of STAT1 in the NK cell response to MCMV infection using STAT1-deficient mice. Equal numbers of WT and Stat1 −/− Ly49H + NK cells were cotransferred into Ly49H-deficient hosts and then infected with MCMV. Similar to Ifnar −/− NK cells, Stat1 −/− NK cells exhibited a striking defect in expansion during the immune response to MCMV infection ( Fig. 2 A) , even though Stat1 −/− NK cells were able to mature nearly as well as WT NK cells after infection (Fig. 2, B and C) . These data support the importance of STAT1-mediated type I IFN signaling in NK cells for an optimal antiviral NK cell response.

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