Selected article for: "endothelial dysfunction and SAON development"
Author: Xie, Xin-Hui; Wang, Xin-Luan; Yang, Hui-Lin; Zhao, De-Wei; Qin, Ling
Title: Steroid-associated osteonecrosis: Epidemiology, pathophysiology, animal model, prevention, and potential treatments (an overview)
  • Document date: 2015_1_13
    • ID: y6y235hw_47
    Snippet: Increased oxidative stress is considered as a crucial cause of SAON. Antioxidative substances such as vitamin E, lipoic acid, and fullerol may alleviate oxidative injury following corticosteroid administration, and thus prevent ON. Vitamin E is a fat-soluble substance in the body that significantly inhibited steroid-induced oxidative stress [49] . Lipoic acid can prevent the development of SAON by inhibiting oxidative stress and amendment of vasc.....
    Document: Increased oxidative stress is considered as a crucial cause of SAON. Antioxidative substances such as vitamin E, lipoic acid, and fullerol may alleviate oxidative injury following corticosteroid administration, and thus prevent ON. Vitamin E is a fat-soluble substance in the body that significantly inhibited steroid-induced oxidative stress [49] . Lipoic acid can prevent the development of SAON by inhibiting oxidative stress and amendment of vascular endothelial dysfunction [50] . Fullerol inhibits adipogenesis and simultaneously enhances osteogenesis by marrow mesenchymal stem cells, possibly through elimination of cellular reactive oxygen species [58] .

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