Title: Research Communications of the 27(th) ECVIM-CA Congress: Intercontinental, Saint Julian's, Malta, 14th to 16th September 2017 Document date: 2017_11_7
ID: roslkxeq_363
Snippet: Disclosures: No disclosures to report. The aim of this study was to i) determine whether there is evidence of CPPV1 infection in feral carnivores and viral traffic between these and companion animals (ii) model in silico the susceptibility of the Tasmanian devil to CPPV1. Reticuloendothelial tissue samples were sourced from deceased feral carnivores for PCR amplification and sequencing of a 529 bp region of the CPPV1 VP2 gene. Phylogenetic analys.....
Document: Disclosures: No disclosures to report. The aim of this study was to i) determine whether there is evidence of CPPV1 infection in feral carnivores and viral traffic between these and companion animals (ii) model in silico the susceptibility of the Tasmanian devil to CPPV1. Reticuloendothelial tissue samples were sourced from deceased feral carnivores for PCR amplification and sequencing of a 529 bp region of the CPPV1 VP2 gene. Phylogenetic analysis of partial VP2 gene sequences was performed using the maximum likelihood method. Tasmanian devil susceptibility to infection was investigated by comparison of their transferrin receptor type 2 (TfR) sequence at binding sites for CPPV1 to susceptible carnivores, and phylogenetic analysis of TfR protein.
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