Author: Lu Lu; Liam Brierley; Gail Robertson; Feifei Zhang; Samantha Lycett; Donald Smith; Margo Chase-Topping; Peter Simmonds; Mark Woolhouse
Title: Evolutionary origins of epidemic potential among human RNA viruses Document date: 2019_9_18
ID: 42twx4gm_8
Snippet: The copyright holder for this preprint (which was not peer-reviewed) is the . https://doi.org/10.1101/771394 doi: bioRxiv preprint of HIV-1 and HIV-2) are most likely have evolved through L1-L2-L3/4 transitions rather than 168 L1-L3/4 ( Figure S11 ). 6 . We also estimate that diversification within L3/4 lineages has 177 contributed significantly to human RNA virus diversity, accounting for 30% of species ( Figure 178 1). However, a larger fract.....
Document: The copyright holder for this preprint (which was not peer-reviewed) is the . https://doi.org/10.1101/771394 doi: bioRxiv preprint of HIV-1 and HIV-2) are most likely have evolved through L1-L2-L3/4 transitions rather than 168 L1-L3/4 ( Figure S11 ). 6 . We also estimate that diversification within L3/4 lineages has 177 contributed significantly to human RNA virus diversity, accounting for 30% of species ( Figure 178 1). However, a larger fraction, at least 51%, has been generated directly from L1 lineages. Table S3 ). We have previously suggested an alternative model where the L2 trait is easily 186 evolved and easily lost 3 , but this model is not supported by the analysis reported here.
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