Selected article for: "clinical severity and IFN ligand"

Author: Chin-Yi Chu; Xing Qiu; Matthew N. McCall; Lu Wang; Anthony Corbett; Jeanne Holden-Wiltse; Christopher Slaunwhite; Qian Wang; Christopher Anderson; Alex Grier; Steven R. Gill; Gloria S. Pryhuber; Ann R. Falsey; David J. Topham; Mary T. Caserta; Edward E. Walsh; Thomas J Mariani
Title: Insufficiency in airway interferon activation defines clinical severity to infant RSV infection
  • Document date: 2019_5_20
  • ID: bx49tbui_83
    Snippet: We tested the effects of gain-and loss-of-function manipulation of IFN activity during RSV infection of PHLE cells (Fig 4B) . As expected, pretreatment of PHLE cells with type I (Figure 4B , C) and type III (Supplemental Figure 13 Figure 14) . Critically, but not surprisingly, RUX potentiated viral infection but did not block IFN ligand production (Fig. 4C) . These data indicate IFN signaling is necessary and sufficient to suppress the expression.....
    Document: We tested the effects of gain-and loss-of-function manipulation of IFN activity during RSV infection of PHLE cells (Fig 4B) . As expected, pretreatment of PHLE cells with type I (Figure 4B , C) and type III (Supplemental Figure 13 Figure 14) . Critically, but not surprisingly, RUX potentiated viral infection but did not block IFN ligand production (Fig. 4C) . These data indicate IFN signaling is necessary and sufficient to suppress the expression of correlates of RSVinfected infant clinical severity in primary cultures of pediatric lung epithelial cells.

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