Author: Michael M. Lutz; Megan P. Worth; Meleana M. Hinchman; John S. L. Parker; Emily D. Ledgerwood
Title: Mammalian orthoreovirus infection is enhanced in cells pre-treated with sodium arsenite Document date: 2019_2_20
ID: cpfwzudq_42
Snippet: salubrinal was not anti-viral to T3D reovirus is not unique. Rotavirus, a fellow member of 492 the Reoviride family, as well as other viruses including hepatitis C virus and mouse 493 hepatitis coronavirus, demonstrate continued viral protein synthesis despite heightened 494 eIF2α-phosphorylation [47] [48] [49] . While this provides additional evidence that reovirus 495 benefits from the ISR, it is important to note that reovirus-induced express.....
Document: salubrinal was not anti-viral to T3D reovirus is not unique. Rotavirus, a fellow member of 492 the Reoviride family, as well as other viruses including hepatitis C virus and mouse 493 hepatitis coronavirus, demonstrate continued viral protein synthesis despite heightened 494 eIF2α-phosphorylation [47] [48] [49] . While this provides additional evidence that reovirus 495 benefits from the ISR, it is important to note that reovirus-induced expression of stress 496 response genes is not uniform and that host shutoff strains, including C8 and C87 (Type 3 497 Abney, T3A), induce increased expression of these genes compared to T3D, which is not 498 considered a host shutoff strain. While all reovirus strains tested, including T3A, 499 benefited from SA-pre-treatment, host shutoff strains may respond differently from T3D 500 to salubrinal treatment [15] . 501
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