Author: Henrique Agra Cavalcante-Silva, Luiz; Cristina Madruga Carvalho, Deyse; de Almeida Lima, Éssia; Galvão, José G.F.M.; de França da Silva, Juliane S.; de Sales-Neto, José M.; Rodrigues-Mascarenhas, Sandra
Title: Neutrophils and COVID-19: The road so far Cord-id: vzqeul71 Document date: 2020_11_30
ID: vzqeul71
Snippet: The SARS-Cov2 infection triggers a multisystem inflammatory disorder, knowing as COVID-19, a pandemic disease. This disease is characterized by acute respiratory distress syndrome, cytokine-driven hyperinflammation, and leukocytes count changes. The innate immune response has been linked to COVID-19 immunopathogenesis (e.g., dysfunctional IFN response and myeloid inflammation). In this regard, neutrophils have been highlighted as essential effector cells in the development of COVID-19. This revi
Document: The SARS-Cov2 infection triggers a multisystem inflammatory disorder, knowing as COVID-19, a pandemic disease. This disease is characterized by acute respiratory distress syndrome, cytokine-driven hyperinflammation, and leukocytes count changes. The innate immune response has been linked to COVID-19 immunopathogenesis (e.g., dysfunctional IFN response and myeloid inflammation). In this regard, neutrophils have been highlighted as essential effector cells in the development of COVID-19. This review summarized the significant finds about neutrophils and its effector mechanisms (e.g., neutrophils enzymes and cytokines, neutrophil extracellular traps) in COVID-19 so far.
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