Author: Kaltschmidt, Barbara; Fitzek, Antonia D.E.; Schaedler, Julia; Förster, Christine; Kaltschmidt, Christian; Hansen, Thorsten; Steinfurth, Fabian; Windmöller, Beatrice A.; Pilger, Christian; Kong, Cihang; Singh, Kashika; Nierhaus, Axel; Wichmann, Dominic; Sperhake, Jan; Püschel, Klaus; Huser, Thomas; Krüger, Martin; Robson, Simon C.; Wilkens, Ludwig; Schulte am Esch, Jan
Title: Hepatic vasculopathy and regenerative responses of the liver in fatal cases of COVID-19 Cord-id: 18l906wl Document date: 2021_1_29
ID: 18l906wl
Snippet: Severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2) infects the nasopharynx and lungs and causes coronavirus disease-2019 (COVID-19). It may impact the heart, brain, kidney, and liver.1 Although functional impairment of the liver has been correlated with worse clinical outcomes, little is known about the pathophysiology of hepatic injury and repair in COVID-19.2,3 Histologic evaluation has been limited to small numbers of COVID-19 cases with no control subjects2,4 and demonstrated large
Document: Severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2) infects the nasopharynx and lungs and causes coronavirus disease-2019 (COVID-19). It may impact the heart, brain, kidney, and liver.1 Although functional impairment of the liver has been correlated with worse clinical outcomes, little is known about the pathophysiology of hepatic injury and repair in COVID-19.2,3 Histologic evaluation has been limited to small numbers of COVID-19 cases with no control subjects2,4 and demonstrated largely heterogeneous patterns of pathology.2,3.
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