Author: Jillian N Whelan; Joshua Hatterschide; David M. Renner; Beihua Dong; Robert H Silverman; Susan R Weiss
Title: The host antiviral ribonuclease L protein supports Zika virus replication factory formation to enhance infectious virus production Document date: 2019_11_24
ID: f9mpkzni_22
Snippet: RNase L mediated antiviral activity ( Figure 3D ). These results from cells lacking OAS3 209 suggest that ZIKV infection may utilize an inactive form of RNase L in a unique manner 210 that results in higher virus production than with RNase L absent. revealed that deletion of RNase L did not alter intensity of genome expression when 230 All rights reserved. No reuse allowed without permission......
Document: RNase L mediated antiviral activity ( Figure 3D ). These results from cells lacking OAS3 209 suggest that ZIKV infection may utilize an inactive form of RNase L in a unique manner 210 that results in higher virus production than with RNase L absent. revealed that deletion of RNase L did not alter intensity of genome expression when 230 All rights reserved. No reuse allowed without permission.
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