Author: Hazel Stewart; Katherine Brown; Adam M. Dinan; Nerea Irigoyen; Eric J. Snijder; Andrew E. Firth
Title: The transcriptional and translational landscape of equine torovirus Document date: 2018_4_7
ID: mozfm5ds_51
Snippet: Similarly, whether the initial role of the TRS motif was to merely stimulate the 437 attenuation of RNA synthesis or to direct the discontinuous mechanism is not 438 known. Our data suggests that transcription mechanisms in the Nidovirales fall into 439 multiple categories, each requiring a distinct role of the TRS: (i) homology-driven 440 reinitiation (canonical discontinuous RNA synthesis, as seen in coronaviruses and 441 arteriviruses and to a.....
Document: Similarly, whether the initial role of the TRS motif was to merely stimulate the 437 attenuation of RNA synthesis or to direct the discontinuous mechanism is not 438 known. Our data suggests that transcription mechanisms in the Nidovirales fall into 439 multiple categories, each requiring a distinct role of the TRS: (i) homology-driven 440 reinitiation (canonical discontinuous RNA synthesis, as seen in coronaviruses and 441 arteriviruses and to a low extent, EToV N protein-coding mRNAs); (ii) structure- The copyright holder for this preprint (which was not peer-reviewed) is the . https://doi.org/10.1101/296996 doi: bioRxiv preprint preferentially translated in infected cells; these have been previously identified as 464 interaction partners of arteriviral non-structural protein 1β and contribute to viral 465 RNA replication (41). It therefore appears that torovirus infection induces a similar 466 host response to many nidovirids. 467
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