Author: Almocera, Alexis Erich S.; Quiroz, Griselda; Hernandez-Vargas, Esteban A.
Title: Stability Analysis in COVID-19 Within-Host Model with Immune Response Cord-id: k7jymvpe Document date: 2020_11_3
ID: k7jymvpe
Snippet: The 2019 coronavirus disease (COVID-19) is now a global pandemic. Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) is the causative pathogen of COVID-19. Here, we study an in-host model that highlights the effector T cell response to SARS-CoV-2. The stability of a unique positive equilibrium point, with viral load [Formula: see text] suggests that the virus may replicate fast enough to overcome T cell response and cause infection. This overcoming is the bifurcation point, near which
Document: The 2019 coronavirus disease (COVID-19) is now a global pandemic. Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) is the causative pathogen of COVID-19. Here, we study an in-host model that highlights the effector T cell response to SARS-CoV-2. The stability of a unique positive equilibrium point, with viral load [Formula: see text] suggests that the virus may replicate fast enough to overcome T cell response and cause infection. This overcoming is the bifurcation point, near which the orders of magnitude for [Formula: see text] can be sensitive to numerical changes in the parameter values. Our work offers a mathematical insight into how SARS-CoV-2 causes the disease.
Search related documents:
Co phrase search for related documents, hyperlinks ordered by date