Selected article for: "apoptotic protein and ndv infection"
Author: Yanrong Li; Ying Liao; Qiaona Niu; Feng Gu; Yingjie Sun; Chunchun Meng; Lei Tan; Cuiping Song; Xusheng Qiu; Chan Ding
Title: CHOP and IRE1a-XBP1/JNK signaling promote Newcastle Disease Virus induced apoptosis and benefit virus proliferation
  • Document date: 2018_4_12
    • ID: mrpj8l3q_5
    Snippet: CHOP promotes NDV-induced apoptosis by reducing the level of anti-apoptotic 342 protein BCL-2 and MCL-1 343 We have reported that NDV infection induces both intrinsic and extrinsic 344 apoptosis, and the extrinsic apoptosis is mediated by induction of death ligands, such 345 as TNF-α and TRAIL (44). Whether CHOP is involved in NDV-induced intrinsic 346 apoptosis? It has been known that CHOP promotes mitochondria mediated apoptosis 347 via down-r.....
    Document: CHOP promotes NDV-induced apoptosis by reducing the level of anti-apoptotic 342 protein BCL-2 and MCL-1 343 We have reported that NDV infection induces both intrinsic and extrinsic 344 apoptosis, and the extrinsic apoptosis is mediated by induction of death ligands, such 345 as TNF-α and TRAIL (44). Whether CHOP is involved in NDV-induced intrinsic 346 apoptosis? It has been known that CHOP promotes mitochondria mediated apoptosis 347 via down-regulation of the pro-survival BCL-2 family (24, 51). Thus, it will be 348 interesting to check whether the expression level of BCL-2 family is regulated by 349 NDV infection. Western blotting analysis showed that pro-survival BCL-2 and 350 MCL-1 were gradually decreased by 0 to 0.5-fold from 16 to 24 h.p.i in NDV-infected 351 HeLa cells ( Fig. 2A) . However, BCL-xL remained relatively stable along the infection 352 author/funder. All rights reserved. No reuse allowed without permission.

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