Author: Gloria P. Larson; Vy Tran; Shuiqìng Yú; Yíngyún Caì; Christina A. Higgins; Danielle M. Smith; Steven F. Baker; Sheli R. Radoshitzky; Jens H. Kuhn; Andrew Mehle
Title: EPS8 facilitates uncoating of influenza A virus Document date: 2019_3_28
ID: muq5rkaa_11
Snippet: Notably, some of our strongest hits for suppressors of FLUAV infection were the interferon-inducible transmembrane proteins (IFITMs). IFITM1, IFITM2, and IFITM3 have previously been characterized as potent inhibitors of FLUAV infection, providing confidence in our approach ( Figure 1D ) (Brass et al., 2009) . Most other candidate genes, including EPS8, have not been previously associated with FLUAV susceptibility, revealing that gene correlation .....
Document: Notably, some of our strongest hits for suppressors of FLUAV infection were the interferon-inducible transmembrane proteins (IFITMs). IFITM1, IFITM2, and IFITM3 have previously been characterized as potent inhibitors of FLUAV infection, providing confidence in our approach ( Figure 1D ) (Brass et al., 2009) . Most other candidate genes, including EPS8, have not been previously associated with FLUAV susceptibility, revealing that gene correlation analysis can identify new host factors that regulate FLUAV infection.
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