Author: Kwang Su Kim; Keisuke Ejima; Yusuke Ito; Shoya Iwanami; Hirofumi Ohashi; Yoshiki Koizumi; Yusuke Asai; Shinji Nakaoka; Koichi Watashi; Robin N Thompson; Shingo Iwami
Title: Modelling SARS-CoV-2 Dynamics: Implications for Therapy Document date: 2020_3_27
ID: 09r4d3nu_14
Snippet: Another possible antiviral mechanism is cytotoxic effects by adaptive 171 immunity including those mediated by cytotoxic T lymphocytes. Here, we assume 172 that promoting cytotoxicity increases the virus death rate by at most two times (i.e., 173 0.1 ≤ ≤ 1), that is, achieves up to 50% reduction of the mean length of virus 174 production. Compared with the other two therapies (blocking de novo infection and 175.....
Document: Another possible antiviral mechanism is cytotoxic effects by adaptive 171 immunity including those mediated by cytotoxic T lymphocytes. Here, we assume 172 that promoting cytotoxicity increases the virus death rate by at most two times (i.e., 173 0.1 ≤ ≤ 1), that is, achieves up to 50% reduction of the mean length of virus 174 production. Compared with the other two therapies (blocking de novo infection and 175
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