Author: Labrie, Marilyne; Lalonde, Simon; Najyb, Ouafa; Thiery, Maxime; Daneault, Caroline; Des Rosiers, Chrisitne; Rassart, Eric; Mounier, Catherine
Title: Apolipoprotein D Transgenic Mice Develop Hepatic Steatosis through Activation of PPAR? and Fatty Acid Uptake Document date: 2015_6_17
ID: 0wtq1c15_44
Snippet: It is also unlikely that our observations are due to an insertion of the apoD transgene in a genomic area disrupting the metabolism, since we observed a similar phenotype in Tg mice where H-apoD expression was driven by the neuron-specific enolase promoter (NSE) [15] . The behavioral, molecular, biochemical and general health characterization demonstrated similar phenotypes between the NSE and the Thy-1 strains. However, we only studied the Thy-1.....
Document: It is also unlikely that our observations are due to an insertion of the apoD transgene in a genomic area disrupting the metabolism, since we observed a similar phenotype in Tg mice where H-apoD expression was driven by the neuron-specific enolase promoter (NSE) [15] . The behavioral, molecular, biochemical and general health characterization demonstrated similar phenotypes between the NSE and the Thy-1 strains. However, we only studied the Thy-1 mice because the hepatic steatosis was more pronounced in this line.
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