Author: Hermesh, Tamar; Moltedo, Bruno; López, Carolina B.; Moran, Thomas M.
Title: Buying Time—The Immune System Determinants of the Incubation Period to Respiratory Viruses Document date: 2010_11_18
ID: 0yqv2osb_53
Snippet: Studies tracking DC migration from the lung to the MLNs during influenza infection using fluorescent reagents that induce unspecific inflammation have shown that DCs migrate from the lung to the MLNs rapidly [127] [128] [129] . It is likely that the viral antagonist is unable to inhibit the inflammation triggered by these inflammation inducing fluorescent reagents. However, when no inflammatory agent is present in the tracking reagent, the kineti.....
Document: Studies tracking DC migration from the lung to the MLNs during influenza infection using fluorescent reagents that induce unspecific inflammation have shown that DCs migrate from the lung to the MLNs rapidly [127] [128] [129] . It is likely that the viral antagonist is unable to inhibit the inflammation triggered by these inflammation inducing fluorescent reagents. However, when no inflammatory agent is present in the tracking reagent, the kinetics of DC migration from the lung to the MLNs during influenza virus infection is quite slow and correlates with the termination of the "stealth phase". DC migration begins around two days after infection when small numbers of DCs carrying viral antigens are first seen in the MLNs and reach a plateau around 3-4 days after infection [96, 122, 124, 130] . Therefore, inhibiting inflammation for two days not only affects innate immunity but also delays the initiation of adaptive immunity.
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