Author: Manandhar, Bandana; Paudel, Pradeep; Seong, Su Hui; Jung, Hyun Ah; Choi, Jae Sue
Title: Characterizing Eckol as a Therapeutic Aid: A Systematic Review Document date: 2019_6_18
ID: 0dpv85od_142
Snippet: The excessive generation of ROS causing oxidative stress in the skin is due to the UVB radiation ultimately damaging DNA, leading to cancer [116, 117] . According to studies on skin cells, it has been demonstrated that the damage of molecules and alteration of their structures, resulting in changes in cellular functions is also due to UVB radiation. Insights were reported about eckol and its ability to protect against UVB-induced oxidative cell d.....
Document: The excessive generation of ROS causing oxidative stress in the skin is due to the UVB radiation ultimately damaging DNA, leading to cancer [116, 117] . According to studies on skin cells, it has been demonstrated that the damage of molecules and alteration of their structures, resulting in changes in cellular functions is also due to UVB radiation. Insights were reported about eckol and its ability to protect against UVB-induced oxidative cell damage in human skin keratinocytes (HaCaT). Eckol decreased the UVB-induced levels of intracellular ROS eventually lowering the damage to cellular components caused by oxidative stress. The cell viability increased by 16% in UVB-irradiated cells treated with eckol (83%) as compared to UVB irradiated cells without eckol (67%) [58] .
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