Selected article for: "autophagy induction and liver level"
Author: Kanasaki, Keizo; Kawakita, Emi; Koya, Daisuke
Title: Relevance of Autophagy Induction by Gastrointestinal Hormones: Focus on the Incretin-Based Drug Target and Glucagon
  • Document date: 2019_5_16
    • ID: 1s44e2le_31
    Snippet: in patients who underwent resection of parts of the stomach, in which the gastrojejunostomy bypasses the pylorus. Therefore, induced levels of GLP-1 during an oral glucose or meal test have been consistently reported after RYGB (Yi et al., 2008) , BPD (Jin and Weng, 2016) , and SG (Moellmann et al., 2018) (Figure 4) . Even though none of these theories have been mechanistically proven during a hypoglycemic event, an increase in GLP-1 could be ass.....
    Document: in patients who underwent resection of parts of the stomach, in which the gastrojejunostomy bypasses the pylorus. Therefore, induced levels of GLP-1 during an oral glucose or meal test have been consistently reported after RYGB (Yi et al., 2008) , BPD (Jin and Weng, 2016) , and SG (Moellmann et al., 2018) (Figure 4) . Even though none of these theories have been mechanistically proven during a hypoglycemic event, an increase in GLP-1 could be associated with hepatic autophagy. He et al. (2015) reported that in a study of rats with obesity and diabetes, RYGB led to significant induction of autophagy in the liver, restored autophagy levels in the liver and was associated with reducing the level of hepatic lipids. Increased autophagy in the liver after RYGB was well correlated with plasma GLP-1 levels . Therefore, induction of GLP-1 after bariatric surgery would also lead to significant stimulation of autophagy (Figure 4) .

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