Selected article for: "previous study and study infection"
Author: Qian, Shaoju; Gao, Zitong; Cao, Rui; Yang, Kang; Cui, Yijie; Li, Shaowen; Meng, Xianrong; He, Qigai; Li, Zili
Title: Transmissible Gastroenteritis Virus Infection Up-Regulates FcRn Expression via Nucleocapsid Protein and Secretion of TGF-ß in Porcine Intestinal Epithelial Cells
  • Document date: 2020_1_21
    • ID: 06qddkw0_38
    Snippet: The secretory IgA and IgG play major roles in mucosal antiinfection immunity and are obtained primarily through the polymeric immunoglobulin receptor (plgR) and FcRn-mediated transport. Reovirus, dsRNA (a ligand for TLR3), LPS (a ligand for TLR4), IFN-γ, IL-4, IL-1β, TNF-α, and TGF-β all regulate pIgR expression (Johansen and Brandtzaeg, 2004; Pal et al., 2005; Schneeman et al., 2005) . Therefore, LPS, IL-1β, and TNF-α could up-regulate the.....
    Document: The secretory IgA and IgG play major roles in mucosal antiinfection immunity and are obtained primarily through the polymeric immunoglobulin receptor (plgR) and FcRn-mediated transport. Reovirus, dsRNA (a ligand for TLR3), LPS (a ligand for TLR4), IFN-γ, IL-4, IL-1β, TNF-α, and TGF-β all regulate pIgR expression (Johansen and Brandtzaeg, 2004; Pal et al., 2005; Schneeman et al., 2005) . Therefore, LPS, IL-1β, and TNF-α could up-regulate the expression of FcRn via the NF-κB pathway. NF-κB appears to be a positive regulator of FcRn expression. In our previous study, we identified that TGEV infection up-regulated FcRn via the activation of the NF-κB pathway (Guo et al., 2016b) . Consistent with this, the replication of the virus is directly related to FcRn expression.

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