Author: Hershenson, Marc B.
Title: Rhinovirus-Induced Exacerbations of Asthma and COPD Document date: 2013_2_21
ID: 1kdc6xk8_32
Snippet: The notion that early rhinovirus infection could modulate future immune responses, leading to allergic airway inflammation, has been bolstered by the discovery of novel epithelial-derived cytokines which promote Th2 differentiation. These cytokines, thymic stromal lymphopoietin (TSLP), IL-25, and IL-33, play a key role in the maturation of Th2 cells via dendritic cell (DC) activation [133] [134] [135] [136] [137] [138] , as well as the activation.....
Document: The notion that early rhinovirus infection could modulate future immune responses, leading to allergic airway inflammation, has been bolstered by the discovery of novel epithelial-derived cytokines which promote Th2 differentiation. These cytokines, thymic stromal lymphopoietin (TSLP), IL-25, and IL-33, play a key role in the maturation of Th2 cells via dendritic cell (DC) activation [133] [134] [135] [136] [137] [138] , as well as the activation of mast cells and iNKT cells [139] . Moreover, TSLP, IL-25, and IL-33 induce IL-13 production by two novel innate effector leukocytes that mediate Th2 immunity-type 2 innate lymphoid cells (also called natural helper cells or nuocytes) [100] [101] [102] [103] [104] [105] [106] and type 2 myeloid cells [107] .
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