Author: William Rodriguez; Aman Srivastav; Mandy Muller
Title: C19ORF66 broadly escapes viral-induced endonuclease cleavage and restricts Kaposi’s Sarcoma Associated Herpesvirus (KSHV) Document date: 2018_12_26
ID: 5a4i8v59_25
Snippet: The copyright holder for this preprint (which was not peer-reviewed) is the author/funder. . https://doi.org/10.1101/506410 doi: bioRxiv preprint identified escaping transcripts with the hope of understanding the protein pre-requisite to 236 forming a protective complex. More globally, determining whether such RNA elements impact 237 RNA fate in uninfected cells will also be key in deciphering their role. To date, no such RNA 238 elements have be.....
Document: The copyright holder for this preprint (which was not peer-reviewed) is the author/funder. . https://doi.org/10.1101/506410 doi: bioRxiv preprint identified escaping transcripts with the hope of understanding the protein pre-requisite to 236 forming a protective complex. More globally, determining whether such RNA elements impact 237 RNA fate in uninfected cells will also be key in deciphering their role. To date, no such RNA 238 elements have been found in viral genes, suggesting that this could be a cell specific 239 mechanism that has evolved in response to viral infection. 240
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