Title: 2018 ACVIM Forum Research Abstract Program: Seattle, Washington, June 14 - 15, 2018 Document date: 2018_10_25
ID: 60ceejq1_302
Snippet: Twelve cats were owned and 23 cats were shelter-housed. Two shelter-housed cats were diagnosed with hyperthyroidism and excluded from further evaluation. In the remaining 33 cats, baseline tT4 was 25.9nmol/l (17. 2-39.3), post-tT4 was 64.4nmol/l (29.4-102) and tT4 ratio was 2.41 (1.56-3.95 ). There was no significant difference (p > 0.05) in post-tT4 or tT4 ratio between the 3 groups (group 0, n=8; group 1, n=16; group 2, n=9), therefore no effec.....
Document: Twelve cats were owned and 23 cats were shelter-housed. Two shelter-housed cats were diagnosed with hyperthyroidism and excluded from further evaluation. In the remaining 33 cats, baseline tT4 was 25.9nmol/l (17. 2-39.3), post-tT4 was 64.4nmol/l (29.4-102) and tT4 ratio was 2.41 (1.56-3.95 ). There was no significant difference (p > 0.05) in post-tT4 or tT4 ratio between the 3 groups (group 0, n=8; group 1, n=16; group 2, n=9), therefore no effect of mildmoderate illness on the TSH stimulation test results in this study. In conclusion, diabetic cats have erythrocytes with increased protein oxidation byproducts and reduced antioxidant capacity, suggesting increased oxidative stress. Because oxidative status was not ameliorated by treatment, it is possible that oxidative stress is persistent or needs longer treatment duration to decrease. The reason behind reduced TBAR in diabetic cats remains elusive. Lastly, oxidative status is not different between cats with and without remission. Hospital population cats with DKA were enrolled into a randomized, prospective, blinded, clinical trial. Cats with blood glucose (BG) > 300mg/dL, glucosuria, blood pH < 7.35 but > 7.0, and a blood betahydroxybutyrate (BOHB) concentration > 2.0mmol/L were enrolled.
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