Selected article for: "inflammatory response and local inflammatory response"
Author: OHTANI, Akifumi; KUBO, Masahito; SHIMODA, Hiroshi; OHYA, Kenji; IRIBE, Tadashi; OHISHI, Daiki; ENDOH, Daiji; OMATSU, Tsutomu; MIZUTANI, Tetsuya; FUKUSHI, Hideto; MAEDA, Ken
Title: Genetic and antigenic analysis of Chlamydia pecorum strains isolated from calves with diarrhea
  • Document date: 2015_2_27
    • ID: 4itsd2aq_20
    Snippet: In these 2 cases, it is possible that C. pecorum was either the primary pathogen or an exacerbating factor causing diarrhea. On the other hand, it is thought that C. pecorum causes asymptomatic infections [8] . Recently, Poudel et al. [16] reported that asymptomatic endemic C. pecorum infections reduce growth rates in calves by up to 48%. They considered that the mechanism of growth suppression by subclinical chlamydial infection was malabsorptio.....
    Document: In these 2 cases, it is possible that C. pecorum was either the primary pathogen or an exacerbating factor causing diarrhea. On the other hand, it is thought that C. pecorum causes asymptomatic infections [8] . Recently, Poudel et al. [16] reported that asymptomatic endemic C. pecorum infections reduce growth rates in calves by up to 48%. They considered that the mechanism of growth suppression by subclinical chlamydial infection was malabsorption of nutrients due to a local inflammatory response to intestinal mucosal infection. Additionally, despite the absence of clinical signs, chla- mydial infection was associated with reduced serum iron concentrations and lower hematocrit values, and infected calves were leukopenic [17] . Mohamad and Rodolakis [15] reported that the persistence of C. pecorum strains in the intestines and vaginal mucus of ruminants could cause longterm sub-clinical infection which may affect the animal's health. This may explain the poor weight gain observed in case 2 after recovery from diarrhea. Further studies on the pathogenesis of C. pecorum infections are required.

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